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In animal studies clenbuterol hydrochloride is shown to exhibit anabolic activity, obviously an attractive trait to a bodybuilder or athlete. The major question is whether clenbuterol is superior to phentermine in terms of muscle synthesis or to other acesulfame K antagonists. The purpose of this study was to investigate the effect of clenbuterol and phentermine on muscle synthesis. Eight healthy male subjects were divided into groups that received either clenbuterol hydrochloride 15 mg (control; n = 4), phentermine 5 mg (phentermine group; n = 4), or another acesulfame K-antagonist 5 mg (phentermine group; n = 4). The acesulfame K-agonist was added at a subsequent dose of 5 mg. The study contained a double-blind placebo-controlled design. The primary outcome measures were muscle strength and creatine kinase (CK) activity. The primary analysis was repeated at a dose of 5 mg to examine whether the acesulfame K-antagonist had any significance on muscle strength and CK activity. Results from placebo-controlled clinical trials had shown that the acesulfame K-antagonist had similar muscle enhancing activity as phentermine alone at the dose of 5 mg. A 2-way analysis of variance with repeated measures was used to determine effects of an acesulfame K-antagonist on muscle strength, CK activity, and a secondary analysis of variance to examine whether the two doses of acesulfame K-antagonist had comparable muscle augmenting activity. There were no significant differences in muscle strength between the two doses (P > .05). There were no significant differences between the doses (P > .05). In an exploratory study the study was repeated with an acesulfame K antagonists (5 mg, 15 mg, or 30 mg). There were no significant differences in CK activity (P > .05) or muscle strength (P > .05) between the doses of acesulfame K antagonists at any dose (P > .05). Thus, in this study 5 mg of acesulfame K-antagonists did not significantly augment muscle strength, CK activity, or a secondary analysis of variance to confirm the ability of the acesulfame K agonists to enhance muscle strength. Further studies are needed to determine if the pharmacologic mechanisms of the acesulfame K agonists is more effective than what was observed in this study. Similar articles: